Skeletal System
Rickets
Definition- a disease of infants and young children, is in violation of mineral metabolism, especially phosphorus, calcium, leading to frustration proper skeleton formation and function of internal organs and systems. Rickets mostly sick children aged 3 months to 1 year, often on the 2nd and much less for 3-4-year life.
Stages- Grade I (mild) - characterized by poorly marked signs of the nervous and skeletal systems: sweating, anxiety, minor softening of the skull bones, beads clearly expressed.
Grade II (moderate) - developing through 2 months of onset. Violated the general condition of the child are observed moderate changes in the nervous, muscular and skeletal systems, can increase liver and spleen.
Grade III (severe) - affected nervous and skeletal systems major, decreased muscle tone, loose joints, pronounced bone deformities, enlarged liver and spleen. There are disorders of the cardiovascular and respiratory system, gastrointestinal tract.
History- Roman descriptions of individuals with rickets can be found as early as the second century, and in the 1640s, the condition was well documented as a common bone ailment across England. Unfortunately, the scientifically proven cause of rickets was not identified until the 1920s, and by the 1930s, public-health initiatives recommended fortifying milk with vitamin D and cod-liver oil as a nutritional supplement for young infants and children. This led to a near eradication of rickets in the United States and other industrialized nations. Unfortunately, rickets has made a comeback and is still common in less-developed nations.
Affected population- Moreover, for a variety of reasons, rickets is seen more frequently amongst infants and children living in industrialized nations.
Symptoms-Symptoms include delayed growth, bow legs, weakness, and pain in the spine, pelvis, and legs./
Diagnosis- Arterial blood gases to determine how much oxygen and carbon dioxide are in the blood, blood tests to measure the levels of calcium and phosphate in the blood, and bone X-rays to check for bone deformities.
Treatment- Adding vitamin D or calcium to the diet, medications, or possibly surgery.
Cause- Lack of vitamin D or calcium, although it can also be caused by a genetic defect or another health condition.
Stages- Grade I (mild) - characterized by poorly marked signs of the nervous and skeletal systems: sweating, anxiety, minor softening of the skull bones, beads clearly expressed.
Grade II (moderate) - developing through 2 months of onset. Violated the general condition of the child are observed moderate changes in the nervous, muscular and skeletal systems, can increase liver and spleen.
Grade III (severe) - affected nervous and skeletal systems major, decreased muscle tone, loose joints, pronounced bone deformities, enlarged liver and spleen. There are disorders of the cardiovascular and respiratory system, gastrointestinal tract.
History- Roman descriptions of individuals with rickets can be found as early as the second century, and in the 1640s, the condition was well documented as a common bone ailment across England. Unfortunately, the scientifically proven cause of rickets was not identified until the 1920s, and by the 1930s, public-health initiatives recommended fortifying milk with vitamin D and cod-liver oil as a nutritional supplement for young infants and children. This led to a near eradication of rickets in the United States and other industrialized nations. Unfortunately, rickets has made a comeback and is still common in less-developed nations.
Affected population- Moreover, for a variety of reasons, rickets is seen more frequently amongst infants and children living in industrialized nations.
Symptoms-Symptoms include delayed growth, bow legs, weakness, and pain in the spine, pelvis, and legs./
Diagnosis- Arterial blood gases to determine how much oxygen and carbon dioxide are in the blood, blood tests to measure the levels of calcium and phosphate in the blood, and bone X-rays to check for bone deformities.
Treatment- Adding vitamin D or calcium to the diet, medications, or possibly surgery.
Cause- Lack of vitamin D or calcium, although it can also be caused by a genetic defect or another health condition.